Maulabar Click to allow Flash. PowerPoint PPT presentation free to view. Getting a grip on strangles: PowerPoint Templates — Are you a PowerPoint presenter looking to impress your audience with professional layouts? User Last modified by: You can use PowerShow. The serological response of foals to vaccination against strangles. History of Patient With Headache.
|Published (Last):||13 May 2009|
|PDF File Size:||5.90 Mb|
|ePub File Size:||9.15 Mb|
|Price:||Free* [*Free Regsitration Required]|
Corresponding author. Address all correspondence and reprint requests to Dr. Abstract A 7-year-old Arabian gelding was presented with a 9-month history of progressive patches of nonpruritic scaling, crusting, alopecia, and leukoderma of the periocular areas and muzzle, becoming generalized over time. Sebaceous adenitis was diagnosed on histopathologic examination. Lesions resolved without treatment, coinciding with regression of a sarcoid on the neck. The lesions began on the periocular and muzzle regions and became generalized to include the face, thorax, abdomen, and rump.
Five months previously, the horse was presented to the Western College of Veterinary Medicine for an unrelated lameness. The horse was otherwise systemically healthy. Dermatologically, there was a crescent-shaped lesion on the margin of the left lower eyelid with mild hyperemia of the conjunctiva and a small 2-cm diameter patch on the right dorsal aspect of the muzzle.
The lesions began to appear approximately 4 mo prior to the initial presentation and had progressed from mildly pruritic patches of crusting and scaling of the hair and skin to nonpruritic fresh, smooth depigmented skin with adherent white scales after the crusts had fallen, or were rubbed, off.
Several punch biopsies 9 mm were taken from the lesions and submitted for histopathologic examination. Overall, the epidermis was normal, apart from lid spongiosis and a progressive or abrupt disappearance of epidermal melanocytes. A mild to moderate mononuclear interface dermatitis was most intense over the areas of depigmentation. Tentative diagnosis at this time was vitiligo Arabian Fading Syndrome and no further treatments were implemented.
All supplements and pellets in the diet had been removed 1 mo prior to presentation to rule out feed allergies, and the horse had been maintained on hay, pasture, and a small amount of oats.
No pasture mates had ever shown similar clinical signs. On current presentation, clinical examination proved the horse to be systemically healthy, and results from a complete blood cell count CBC and biochemical profile showed no significant abnormalities.
The sarcoid on the left side of the neck had reduced minimally in size and was ulcerated on the surface. Dermatological lesions now included multifocal nonpruritic patches of crusting, scaling, alopecia, and depigmentation of the face, neck, trunk, and rump. The 9-month-old lesions on the face were now patches of smooth, depigmented skin with fine white adherent scales Figure 1a. The 2- to 3-month-old lesions of the neck, trunk, and rump consisted of patches of scaling and crusting Figure 1b ; beneath the lesions that could be easily epilated off, the skin was smooth, moist, and depigmented, and no exudate was present Figure 1c.
Sebaceous adenitis in a 7-year-old Arabian gelding
VACUNA ADENITIS EQUINA
VAC-SULES ADENITIS EQUINA